Authors Mohammad Ali Taheri, Sara Torabi, Noushin Nabavi, Farid Semsarha
Publication date Apr 16, 2022
Journal Journal of Cosmointel
Volume cover_issue_3_en_US Vol. 1 No. 2
Pages 08 to 21
Publisher Cosmointel Inc.


Alzheimer’s Disease (AD) is one of the most common causes of dementia, imposing large financial and psychological burdens on nations worldwide. Thus, we direly need new treatment strategies or drugs for this disease. The aim of this study is to investigate the effects of a novel non-pharmacological method in the treatment of Alzheimer’s disease, based on employing Taheri Consciousness Fields. These fields function at the level of cellular and mo-lecular processes. In this study, the effects of Faradarmani Consciousness Field (CF) on the AD mouse model (in vivo) and human neuron cell line in vitro were investigated. In this study, we established a human neuron cell culture as well as a traumatic brain injury (TBI) mouse model. We then measured changes in amyloidopathy, tau protein content, microtubule assembly, neu-ronal cell survival, and finally behavior of TBI mice in Elevated Plus Maze un-der treatment of the Faradarmani CF. According to the results, treatment of human neural cells and a mouse model of Alzheimer’s disease by the Faradarmani CF leads to complete survival of neural cell models and elimination of amyloidopathy and tau protein, and remarkable behavioral improvement of the treated TBI mice model in the elevated plus-maze. Based on the re-sults, Faradarmani CF treatment suppresses AD development in the labora-tory models. In this regard, conducting a human clinical study with the aim of introducing a new global complementary and alternative medicine in AD treatment is highly recommended.

Keywords: Alzheimer’s Disease; Faradarmani Consciousness Field; Taheri Consciousness Fields; TBI; mice model; neural cell model


Citation: Taheri, M. A., Torabi, S., Nabavi, N., & Semsarha, F. (2022). Faradarmani Consciousness Field Suppresses Alzheimer’s Disease Development in both in vitro and in vivo Models of the Disease. Journal of Cosmointel1(2), 8–21. Retrieved from